> Once the body is starving for carbohydrates it begins a process to break down fats and even protein into an energy source. This produces toxins, ketones.
> Both [two of the three types of ketone bodies] are 4-carbon molecules that can readily be converted back into acetyl-CoA [which can directly fuel the krebs cycle] by most tissues of the body, with the notable exception of the liver.
Which is not really matching the definition of 'toxins'.
In my mind, saying that ketone bodies are toxins could be suggested in the fact that ketoacidosis is a real threat when a metabolism is affected by T1. But, again, T1 is not T2, and it's not the condition of a healthy metabolism, which is fully capable of handling ketone bodies, even thriving on it (glucose is fine, too, I'm not trying to sell that line).
Oh thanks, I've been T1 since 1982, so I'm not surprised that science has improved. Keto-acidosis is a toxic state for T1 diabetics at least, I'm not sure if T2 can handle them better.
You may want to read some (hopefully) unbiased information on that subject : https://en.wikipedia.org/wiki/Ketone_bodies https://en.wikipedia.org/wiki/Ketogenesis
For example:
> Both [two of the three types of ketone bodies] are 4-carbon molecules that can readily be converted back into acetyl-CoA [which can directly fuel the krebs cycle] by most tissues of the body, with the notable exception of the liver.
Which is not really matching the definition of 'toxins'.
In my mind, saying that ketone bodies are toxins could be suggested in the fact that ketoacidosis is a real threat when a metabolism is affected by T1. But, again, T1 is not T2, and it's not the condition of a healthy metabolism, which is fully capable of handling ketone bodies, even thriving on it (glucose is fine, too, I'm not trying to sell that line).